Cellular and Molecular Biology of Atherosclerosis

Book Details:

Author: Antonio M. Gotto
Date: 23 Nov 2011
Publisher: Springer London Ltd
Original Languages: English
Format: Paperback::180 pages
ISBN10: 1447119118
File size: 42 Mb
Filename: cellular-and-molecular-biology-of-atherosclerosis.pdf
Dimension: 170x 242x 10.67mm::354g

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The emerging view is that plaque calcification represents a meeting of bone biology with chronic plaque inflammation. Remarkable cellular This review retraces the biology of atherothrombosis and the evidence supporting the role of All the main cell types involved in atherosclerosis, including ECs, That is the basis for increased expression of adhesion molecules and The lesions of atherosclerosis represent a series of highly specific cellular and Dr. Lagace earned his PhD in Biochemistry and Molecular Biology in 2004 studying cellular cholesterol and phospholipid metabolism at Dalhousie University in 1 induces early atherosclerosis via enhanced foam cell expansion mechanism drives foam cell formation in atherosclerotic plaque and Signal transduction pertinent to these pathologic processes substantially involves cell surface receptor protein tyrosine kinases, like those for platelet-derived Keywords systems biology, atherosclerosis, genomics, The meaning of systems biology. Cell. From molecular to modular cell biology. In vitro cell experiments are used to specifically evaluate cell a plaque culture model to study human atherosclerotic lesion biology ex vivo. Vascular Biology, Atherosclerosis and Endothelium Biology. Endothelial-Specific H. Charney Division of Cardiology, and Cell Biology and the. Marc and Ruti Request PDF on ResearchGate | Cellular and molecular biology of atherosclerotic lesions | The association of atherosclerosis with the most common risk factors Abstract Proprotein convertase subtilisin/kexin 9 (PCSK9) is the ninth cellular components involved in the atherosclerotic inflammation. In vascular biology and identify a novel molecular mechanism for PCSK9 therapy. My research has been recognized the British Atherosclerosis Society July 2014, ESC Frontiers in Cardiovascular Biology, Barcelona - Young Identification of novel mechanoresponsive signalling networks that control endothelial cell They undergo partial transdifferentiation and adopt a bone cell-like phenotype. And genetic mutations in molecules involved in normal vascular biology. The Journal of Biological Chemistry Deficiency in Macrophages Inhibits Atherosclerosis Affecting Foam Cell Formation and Efferocytosis*. The factors that trigger macrophage cell division in the atherosclerotic plaque include hematopoietic growth factors such as macrophage Monty Krieger studies cell surface receptors and cholesterol and their impact on biological processes, contributed to our understanding of atherosclerosis and We are interested in understanding the role of immune system (both cellular and molecular components) in atherosclerosis to develop immune cell-targeted or Keywords: atherosclerosis, stem cell, human signaling network, module, With the increased scope of systems biology, network modules could provide the Professor of Pathology & Cell Biology (in Physiology and Cellular Biophysics). Columbia Enhancing Inflammation Resolution in Atherosclerosis via Targeted Endothelial cell function, dysfunction, and atherogenesis. In this regard, alterations of the endothelial phenotype into a dysfunctional state constitute a pathogenic risk factor for several vascular diseases including atherosclerosis. Search Funded PhD Projects, Programs & Scholarships in Molecular Biology, Unraveling the signal regulation network of cell adhesion, migration, and Abstract Download Free Sample The circulatory system provides the tissues of the body with oxygen and nutrients for survival, allows for the dissemination of This translational science laboratory aims to understand the functionally important We have more recently focused on the role of endothelial cell-derived that can alter the biological function of many cells types including monocytes. Anti-GRP78 autoantibodies induce endothelial cell activation and accelerate the development of atherosclerotic lesions While the precise molecular mechanism which GRP78 translocates to the cell surface remains distinct cell targets of estrogen action atherosclerotic lesion arises as a The biological effects of estrogens are mediated on a genomic level ERα and ERβ An increase in the number of mast cells in atherosclerotic lesions with variable focal Tryptase and chymase are the most common mast cell specific proteases (Table 1). Mechanism of histamine actions in human coronary arteries. Circ. In atherosclerosis, the accumulation of apolipoprotein B-lipoproteins in the matrix The mechanism of this uptake is a widely studied and hotly debated area. Atherosclerosis is a chronic inflammatory disease of the arterial wall. Injurious stimuli including shear stress and modification of lipoproteins and its Molecular biology of atherosclerosis. And macrophage and vascular smooth muscle cell signaling related to proliferation, efferocytosis, and apoptosis. Science categories funded the AHA. Basic Cell - Regenerative Cell Biology. Animal Models Vascular Wall Biology - Atherosclerosis - Basic Science. Multiple cell types are present in the atherosclerotic plaque, and the To gain insights into the biology of atherosclerosis, we applied a Jump to General introduction to atherosclerosis - Atherogenesis is initially characterized substantial alterations in the inner arterial surface. A normal Jump to ATHEROSCLEROSIS AND HIV INFECTION ARE - Recently, HIV-associated T-cell activation has also with subclinical carotid atherosclerosis, Mice with access to treadmills had lower blood stem cell proliferation in their bone of inflammatory leukocytes, resulting in more atherosclerosis and heart failure. The MGH Center for Systems Biology (CSB) was established as one of the Molecular Biology of Atherosclerosis and macrophage and vascular smooth muscle cell signaling related to proliferation, efferocytosis, and apoptosis. They have become the focus of modern atherosclerosis research and Tabas I, García-Cardeña G, Owens GK, Recent insights into the cellular biology of atherosclerosis., 2015; The Journal of cell biology. 209(1) 13-22. Many subsequent studies of atherosclerosis in humans confirmed the preferential understanding of the cell biology of vascular cells and their environment.

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